Claudia Sanmiguel，MD

Claudia Sanmiguel, MD

Director, Ingestive Behavior and Obesity Program, Oppenheimer Center for Neurobiology of Stress; Division of Digestive Diseases, David Geffen School of Medicine at UCLA

Claudia Sanmiguel is the Ingestive Behaviors and Obesity Program (IBOP) director at the Oppenheimer Center for Neurobiology of Stress (CNS) and she is a clinical instructor at the UCLA Digestive Diseases Division.She was born in Bogota, Colombia where she studied Medicine at the Pontificia Universidad Javeriana and specialized in Internal Medicine and Gastroenterology.Then she moved to Alberta, Canada where she did research on gastrointestinal motility and the use of artificial pacemakers and stimulators for the treatment of gastrointestinal disorders.She continued her research career at the Cleveland Clinic in Ohio and at the Cedars Sinai Medical Center in Los Angeles, where she explored the use of pacemakers and electrical stimulators for the treatment of obesity and obesity related diabetes mellitus, as well as, studied gastric electromechanical signals related to eating behavior and satiety.As part of pursuing a research career in United States, she completed her residency in Internal Medicine at Cedars-Sinai Medical Center and trained in Gastroenterology at the University of California Los Angeles.She has continued her pursue on understanding the mechanisms that regulate eating behavior in health and in obesity, and the role of the brain/gut/microbiome axis in interpreting and regulating those behaviors.She has published several papers in well known GI and bioengineering journals and presented her research results in North American and International meetings.She currently has partial NIH funding for a study on the role of brain activity and changes in eating behavior in weight loss after bariatric surgery and how some peripheral signals coming from visceral fat and gut microbiome may play a role in obesity and weight loss.She is also doing research on neuroplasticity in brain areas related to eating hehaviors in response to neuromodulation.

Sanmiguel C, Gupta A, Mayer EA.Gut Microbiome and Obesity: A Plausible Explanation for Obesity.Curr Obes Rep.2015 Jun;4(2)***

Sanmiguel CP, Gupta A, Labus LS, Coveleskie K, KaragiannidisI , AlaverdyanM, Ashe-McNalley C, Stains J, and others.Adiposity Is Associated With Alterations Within the Brain Reward System in Adult Subjects.Gastroenterology 2015, Vol.148, Issue 4, S

Gupta A, Mayer EA, Sanmiguel CP, Van Horn JD, Woodworth D, Ellingson BM, Fling C, Love A, Tillisch K, Labus JS.Patterns of Brain Structural Connectivity Differentiate Lean from Overweight Subjects.Neuroimage-Clinical, 2015; 7: 506–

Sanmiguel CP, Coveleskie K, Gupta A, Kilpatrick L, Labus J, Ashe-McNalley C, Dutson EP, Mayer EA.Association of abdominal fat with resting state low frequency brain activity in human subjects.Neurogastroenterology & Motility.2013; 25, Suppl 1:14

Sanmiguel CP, Ito Y, Hagiike M, Conklin JL, Lalezari D, Soffer EE.The effect of eating on Lower Esophageal Sphincter electrical activity.American Journal of Physiology Gastrointest Liver Physiology.2009 Apr; 296(4): ***

Sanmiguel CP, Conklin JF, Cunneen SA, Barnett P, Phillips EH, Kipnes M, Pilcher J, Soffer EE.Gastric Electrical Stimulation with the TANTALUS System in Obese Type 2 Diabetes Patients: Effect on Weight and Glycemic Control.J Diabetes Science and Technology.2009; 3:***

Sanmiguel CP，Haddad W，Aviv R，Cunneen SA，Phillips EH，Kapella W，Soffer EE。TANTALUS TM肥胖症系统：影响胃排空固体和生长激素释放肽的血浆水平。Obes Surg。2007; 17：1503-9

Aviv R，Policker S，Brody F，Bitton O，Haddad W，Kliger a，Sanmiguel CP，Soffer EE。狗的胃电和机械活动的昼夜模式。Neurogastroenterol Motil。2008; 20：63-8

Aviv R，Sanmiguel CP，Kliger A，Policker S，Haddad W，Hagiike M，Soffer EE。使用胃电信号进行狗的自动进食检测?法。Neurogastroenterol Motil。2008,20：369-76。

Kirsten Tillisch，MD

Kirsten Tillisch, MD

Director, Mind Body Research Program, Oppenheimer Center for Neurobiology of Stress; Associate Professor, Department of Medicine, David Geffen School of Medicine at UCLA

Dr.Kirsten Tillisch completed her undergraduate work at the Otis Institute of Parsons School of Design, earning a Bachelor of Fine Arts with Honors.She obtained her medical degree from the David Geffen School of Medicine at UCLA and was elected to the medical honor society Alpha Omega Alpha.She continued on at UCLA to complete her training in internal medicine and gastroenterology, graduating in 2003.Her clinical interests are functional bowel disorders such as irritable bowel syndrome, functional dyspepsia, and cyclic vomiting syndrome.Her research interests include brain-gut interactions , the effects of nonpharmacological therapies on functional gastrointestinal disorders, and pharmacological treatment of irritable bowel syndrome.Her recent research projects include defining resting state brain dysfunction in irritable bowel syndrome patients, evaluating the role of gut microbiota modulation on emotional processing in the brain, and assessment of neurokinin-1 receptor antagonists effects on the gut and brain in irritable bowel syndrome.She is a member of the Neuroimaging Program of the Gail and Gerald Oppenheimer Family Center for Neurobiology of Stress.

Mayer EA, Tillisch K, Gupta A.Gut/brain axis and the microbiota.J Clin Invest.2015 Mar 2;125(3):926-38.doi: 10.1172/JCI76304.Epub 2015 Feb 17.Review.PubMed PMID: 25689247; PubMed Central PMCID: PMC***

Mayer EA, Knight R, Mazmanian SK, Cryan JF, Tillisch K.Gut microbes and the brain: paradigm shift in neuroscience.J Neurosci.2014 Nov 12;34(46):15490-6.doi: 10.1523/JNEUROSCI.3299-14.2014.Review.PubMed PMID: 25392516; PubMed Central PMCID: PMC***

Mayer EA, Padua D, Tillisch K.Altered brain-gut axis in autism: comorbidity or causative mechanisms Bioessays.2014 Oct;36(10):933-9.doi: 10.1002/bies.201400075.Epub 2014 Aug 22.Review.PubMed PMID: 2***

Tillisch K，Labus JS。神经影像的微生物 - 肠 - 脑轴。Adv Exp Med Biol。2014; 817：405-16。doi：10.1007 / 978-1-4939-0897-4_18。评论。PubMed PMID：2499***

Tillisch K.肠道微生物群对人类中枢神经系统功能的影响。肠道微生物。2014年5月 - 6月; 5（3）：404-10。doi：10.4161 / gmic.29232。电子书2014年5月16日。审查。PubMed PMID：24838095; PubMed中央PMCID：PMC415***

Tillisch K，Labus J，Kilpatrick L，Jiang Z，Stains J，Ebrat B，Guyonnet D，Legrain-Raspaud S，Trotin B，Naliboff B，Mayer EA。用益生菌消耗发酵乳产品调节大脑活动。消化内科。2013年6月; 144（7）：1394-401,1401.e1-4。doi：10.1053 / j.gastro.2013.02.043。Epub 2013 Mar 6。PubMed PMID：23474283; PubMed中央PMCID：PMC3839572。

Elizabeth Videlock, MD

研究领域肥胖与代表谢

全球范围内肥胖流行率的快速增长与对这种流行病相关机制的重新理解一致。在许多因素中，可能的因果关系中，我们的饮食变化（加工食品消费增加）被认为是最重要的因素之一。由于肠是人体中最密集的殖民地和最多样的微生物群落，并且由于肠道微生物组成和代表谢功能与饮食组成有内在联系，所以人们不得不质疑肠道微生物群与肥胖之间的关系。虽然一些研究松散地关联了肥胖与肠道微生物组成的变化，但这一领域的发现是相互矛盾的。然而，一些重要的研究已经证明，在特定的情况下，从肥胖的供体向微生物小鼠引入微生物群能够“传递”肥胖的型。这些研究对于理解肠道微生物本身能够与宿主相互作用以改变宿主的身体组成是决定性的。大多数研究已经在无菌小鼠中进行。然而，一些流行病学研究已经显示了早期使用抗生素和在生命的头几年发展肥胖的倾向之间的关系。此外，在食品中添加抗生素以促进牲畜生长的老式普遍做法支持微生物对身体组成的影响。

代表谢组学研究的进展为理解微生物与宿主之间的关系打开了一扇新的大门。由消化道细菌产生的代表谢物可以进入我们的血液，事实上，高达三分之一的人类血液中的小分子来自肠道细菌。这些代表谢物中的一些已经与人宿主中的有益作用相关联; 而且这些代表谢物的改变也会导致有害的结果，例如增加炎症和促进胰岛素抵抗和肥胖。

摄食行为与肥胖计划（IBOP），奥本海默应激神经生物学中心（CNS

UCLA CNS摄食行为与肥胖计划研究外围信号（肠道微生物代表谢物，肠道肽，迷走神经和脂肪组织信号传导）与大脑在控制健康和肥胖的体重和饮食行为之间双向交流的机制。近年来，大量证据明肠道微生物群与宿主体重之间存在重要联系。无菌动物研究明，肠道微生物组的变化可以改变饮食行为。这些行为效应的机制尚不清楚，但可能涉及肠道微生物群的信号通过迷走神经，肠道激素或微生物代表谢物传递给大脑。我们正在进行的研究旨在确定这些相互作用的基础。特别，我们旨在描述肠道微生物代表谢物谱如何通过改变与饱食，饥饿和进食行为相关的大脑结构/功能来控制体重。我们还将描述性别和种族/种族对肥胖的肠微生物 - 脑相互作用的影响。

研究领域口腔生物学

分子生物学技术的最新进展扩大了我们对细菌多样性和口腔内物种之间发生的协会群落相互作用的理解。它们导致我们对口腔微生物组及其在健康和疾病方面的理解的巨大进展，不仅与口腔传染病如蛀牙和牙龈疾病有关，而且还与全身性疾病有关。

使用独立于文化的方法进行的研究揭示了居住在口腔内的多种微生物的严重程度。估计人口含有700多种不同的细菌种类，其中包括最复杂的微生物菌群之一。居住在口腔中的微生物的多样性包括细菌，古细菌，螺旋体，原生动物，支原体，酵母和真菌。微生物菌群的多样性反映了遗传信息的巨大多样性和巨大的生物生理潜力。如果我们认为平均细菌种类有2,000-6,000个基因，that 大约700个个体的口腔细菌群代表超过100万个基因的池，比人类宿主基因多10倍。

Drs博士的主要研究兴趣。加州大学洛杉矶分校牙科学院的何文渊教授和薛雪鹤博士主要关注口腔微生物在宿主健康和疾病中的作用，并探讨实际的因果关系。同时，他们也积极研究个别物种的口腔微生物生态学，物种间以及多物种水平。他们的最终目标是更好地了解口腔微生物组的生态学特征，口腔细菌种类的详细特征将成为打击牙科，牙周以及与口腔微生物有关的更具体和更有效的预防和治疗方法的门户全身性疾病。

目前在李惠英博士的口腔微生物组项目该小组着重了解口腔微生物在牙周健康和疾病中的作用。Li实验室通过比较健康人和牙周炎患者龈下菌群宏基因组的差异，旨在确定区分牙周健康和疾病龈下微生物群的微生物群落和宏基因组特征。此外，2型糖尿病与牙周炎的患病率和进展相关。通过比较2型糖尿病患者和非糖尿病患者的牙周微生物群，李实验室正在研究宿主因素（包括免疫应答）对牙周健康和疾病的口腔微生物群的影响。

Guo，L.，JS。McLean，Y.Yang，R.Eckert，CW Kaplan，P.Kyme，O.Sheikh，B.Varnum，R.Lux，W.Shi和X.He 。2015作为人类微生物生态学的靶向调节剂的精确引导的抗微生物肽。Proc Natl Acad Sci USA 112（24）：***

他，X.，JS。McLean，A.Edlund，S.Yooseph，AP Hall，SY。Liu，P.Dorrestein，E.Esquenazi，R.Hunter，G.Cheng，KE。纳尔逊，勒克斯和史密斯。一个人类相关的TM7的驯化揭示了一个减少的基因组和寄生生活方式。Proc Natl Acad Sci USA 112（1）：244-9。PMCID：PM***

Wu，T.，L.Cen，C.Kaplan，X.Zhou，R.Lux，W.Shi和X.Hho。介导白色念珠菌（Candida albican）和有核梭杆菌（Fusobacterium nucleatum）的共聚集的细胞成分。牙科研究杂志 DOI：10.1177 / 00220345***

主要教师

Xuesong He，DDS，PhD。

Xuesong He, DDS., PhD

Assistant Adjunct Professor, UCLA School of Dentistry

UCLA School of Dentistry

20-118 CHS

Los Angeles CA 9***

Phone: (310)***

E-mail: xhe**[ta]**tistry.ucla.edu

Dr.Xuesong He is an Adjunct Assistant Professor in School of Dentistry at UCLA.Dr.He received his D.D.S.from Peking University Health Science Center in 1999, and his PhD in Microbiology from Indiana University in USA in 2006.Dr.He’s research interest includes: 1) Interspecies interaction between oral microbes; 2) culturing and studying”yet-to-be” cultivated oral microbes; and 3) studying the oral microbial ecology and its impact on human oral health and diseases.He is currently the co-principle investigator of a NIH RO1 grant on studying the unique epibiotic parasitic relationship between two oral commensal bacteria that could be involved in oral mucosal infectious disease.The international impact of his research can be proved by his over 30 well-cited publications in peer-reviewed leading scientific journals including: PNAS, ISME Journal, Advance in Dental Research, Microbiology and Molecular Biology Reviews, Journal of dental research, Journal of Endodontics, Molecular Microbiology, Microbiome Journal, Scientific Reports, Frontiers in Microbiology, Molecular Oral Microbiology, Microbial Ecology, Plos One, International Journal of Oral Science, FEMS Microbiology Letter and Journal of Bacteriology, etc.

Guo, L., JS.McLean, Y.Yang, R.Eckert, C.W.Kaplan, P.Kyme, O.Sheikh, B.Varnum, R.Lux, W.Shi and X.He.2015 A precision guided antimicrobial peptide as a targeted modulator of human microbial ecology.Proc Natl Acad Sci USA 112(24): ***

He, X., JS.McLean, A.Edlund, S.Yooseph, A.P.Hall, SY.Liu, P.Dorrestein, E.Esquenazi, R.Hunter, G.Cheng, KE.Nelson, R.Lux and W.Shi.2015.Domestication of a human-associated TM7 reveals a reduced genome and parasitic lifestyle.Proc Natl Acad Sci USA 112(1):244-9.PMCID:PM***

Wu, T., L.Cen, C.Kaplan, X.Zhou, R.Lux, W.Shi and X.He.2015.Cellular components mediating co-aggregation of Candida albican and Fusobacterium nucleatum.Journal of Dental Research.DOI:10.1177/00220345***

资助机构/拨款号码：NIH NIDCR 1R01DE02

标题：“TM7-最难以捉摸的口腔门的驯化和征”

李慧英博士

Huiying Li, PhD

Assistant Professor, Department of Molecular & Medical Pharmacology; Faculty, Crump Institute for Molecular Imaging, UCLA

4339 CNSI

570 Westwood Plaza, Building

洛杉矶 CA 9***

电话：（310***

电话：（310***

电子邮件：huiying**[ta]**net.ucla.edu

网站：Li Lab

My interest in the microbiome research began from The Sorcerer II Global Ocean Sampling Expedition led by the J Craig Venter Institute several years ago, where I applied bioinformatics to study microbial protein families in the ocean microbiome.The current research in my lab focuses on understanding the human microbiome, the collective genome of trillions of microorganisms residing in the human body, and its interactions with the host in relation to human health and diseases.Using multi-disciplinary approaches, including genomics, metagenomics, bioinformatics, high-throughput sequencing, microbiology, and biochemistry, we aim to identify the molecular mechanism of the human microbiome in health and disease pathogenesis and to develop diagnostic markers and therapeutics for microorganism-related human diseases.By combining computational and experimental approaches, the ultimate goal of my research is to understand the human-microbiota symbiotic system at both molecular level and systems level.

My research group is highly experienced in high-throughput sequencing and data analysis with expertise in bioinformatics, 16S rDNA sequence analysis, metagenomic shotgun sequence analysis, RNA-Seq data analysis, and genome assembly, annotation and comparison.I was funded by the Human Microbiome Project (HMP) among the 15 Demonstration Projects to study the role of the human skin microbiome in acne.I am currently funded by the NIGMS and NIDCR to study the human skin microbiome and oral microbiome and their associations with diseases.

Kang D, Shi B, Erfe MC, Craft N, Li H.Vitamin B12 modulates the transcriptome of the skin microbiota in acne pathogenesis.Science Translational Medicine.2015; 293(7): 293ra

Liu J, Yan R, Zhong Q, Ngo S, Bangayan NJ, Nguyen L, Lui T, Liu M, Erfe MC, Craft N, Tomida S, Li H.The Diversity and Host Interactions of Propionibacterium acnes Bacteriophages on Human Skin.The ISME Journal.2015; 9: 2078-2

Shi B, Chang M, Martin J, Mitreva M, Lux R, Klokkevold P, Sodergren E, Weinstock GM, Haake SK, Li H.Dynamic Changes in the Subgingival Microbiome and Their Potential for Diagnosis and Prognosis of Periodontitis.mBio.2015; 6(1): e0***

Liu J, Cheng A, Bangayan NJ, Barnard E, Curd E, Craft N, Li H.Draft Genome Sequences of Propionibacterium acnes Type Strain ATCC6919 and Antibiotic-Resistant Strain HL411PA1.Genome Announcements.2014; 2(4): e0***

Kasimatis G, Fitz-Gibbon S, Tomida S, Wong M, Li H.Analysis of Complete Genomes of Propionibacterium acnes Reveals a Novel Plasmid and Increased Pseudogenes in an Acne Associated Strain.BioMed Research International.2013; 2013:***

Tomida S, Nguyen L, Chiu BH, Liu J, Sodergren E, Weinstock GM, Li H.Pan-Genome and Comparative Genome Analyses of Propionibacterium acnes Reveal Its Genomic Diversity in the Healthy and Diseased Human Skin Microbiome.mBio.2013; 4(3): e0***

Fitz-Gibbon S, Tomida S, Chiu BH, Nguyen L, Du C, Liu M, Elashoff D, Erfe MC, Loncaric A, Kim J, Modlin RL, Miller JF, Sodergren E, Craft N, Weinstock GM, Li H.Propionibacterium acnes Strain Populations in the Human Skin Microbiome Associated with Acne.Journal of Investigative Dermatology.2013; 133: 2152-2

资助机构/拨款号码：NIH R01 GM09

标题：“人类皮肤微生物体中痤疮丙酸杆菌及其噬菌体的种群动态”

资助机构/拨款号码：NIH R01 DE02

标题：“健康和糖尿病患者牙周微生物群的宏基因组学研究”

Wenyuan Shi博士

研究领域皮肤病

皮肤是人体内最大的器官，是抵御环境攻击和病原体入侵的第一道防线。皮肤也是数百种微生物的宿主，包括细菌，真菌，真核细胞螨和病毒。出生后不久，不同皮肤部位的微生物群落不同，具有独特的生理和免疫龛位。它们在感染环境，防止病原体定殖和感染以及引导宿主免疫系统响应外来侵入方面发挥重要作用。驻留的微生物通过皮肤分泌物隔离营养物，并通过微生物群落内的复杂相互作用和宿主与宿主皮肤形成动态生态系统。组成，动态，李实验室

李慧英博士的研究小组中的皮肤微生物组项目着重于了解皮肤微生物在健康和疾病中的分子功能。通过研究生命在人体皮肤上的微生物的基因组和转录组，李实验室研究皮肤微生物如何与宿主相互作用，并在皮肤疾病中起作用。目前正在研究两种最常见的皮肤疾病，寻常痤疮和特应性皮炎中的微生物群，特别是在菌株水平和功能水平。这些研究将提供关于共生皮肤微生物群在健康和疾病中的作用的分子观察，并且将提高我们对涉及微生物群的疾病机制的理解以及微生物群在疾病治疗和皮肤健康维护中的潜在未来操纵。

电话：（310***

电子邮件：bento**[ta]**m.ucla.edu

网站：Laboratory

衰老研究领域

科学和医学的进步使世界各国的预期寿命稳步提高。一些老年人现出对神经变性或感染性疾病的易感性增加，以及由于衰老过程而导致的生理功能正常退化以外的健康逐渐下降。老年人特别有营养不良的风险，可导致功能衰退。肠道微生物群与宿主的健康和疾病密切相关，在人体新陈代表谢，免疫和神经系统功能的发育和调节中起主要作用，对维持心理和生理健康至关重要。抑郁和认知下降不一定是老化的一部分。

Helen Lavretsky博士是加州大学洛杉矶分校（UCLA）的老年精神病学家，研究兴趣在于治疗和预防情绪和认知障碍。她的研究已经明，诸如瑜伽和太极拳这样的身心锻炼可以帮助减少衰老成年人和紧张照顾者的抑郁和认知能力下降。目前的研究提供参与太极和瑜伽研究，以及老年抑郁症和轻度认知功能障碍的药理研究。Lavretsky博士旨在通过比较患有心境和认知症状的患者中的微生物组成，以及在老年人中开发针对预防情绪障碍和认知衰退的微生物和健康的干预措施，来研究微生物在晚期心境和认知障碍中的作用。

Lee SM，Lavretsky H.“微生物和衰老疾病”IN：精神健康和衰老的补充和整合疗法。编辑：Lavretsky，Sajatovic，Reynolds，牛津大学出版社正在出版。

电话：（310***

电子邮件：hlavretsky**[ta]**net.ucla.edu

Dr.Helen Lavretsky is the Geriatric Psychiatrist at UCLA with research interests in treatment and prevention of mood and cognitive disorders of aging.Her studies have already shown that mind-body exercise such as yoga and Tai Chi can help to reduce depression and cognitive decline in aging adults and in stressed caregivers.Current research studies offer participation in Tai Chi and yoga studies, as well as pharmacological studies for geriatric depression and mild cognitive impairment.

Dr.Lavretsky aims to investigate the role of microbiota in late life mood and cognitive disorders by comparing microbiome composition in those with and without mood and cognitive symptoms, and to develop interventions targeting microbiota and wellness for prevention of mood disorders and cognitive decline in older adults

David Walker博士

David Walker, PhD

Professor, Department of Integrative Biology and Physiology, UCLA

微生物核心

主席：Jonathan Jacobs博士（JJacobs**[ta]**net.ucla.edu

经理：Venu Lagishetty博士（VLagishetty**[ta]**net.ucla.edu）

微生物核心位于健康科学大楼中心，A3-115。它对人类和动物样本（包括粪便，组织和肠道灌洗物质）进行16S核糖体RNA基因测序。

结果显示为与Greengenes数据库匹配的操作分类单元的计数。微生物核心还提供了额外的服务，以促进样品采集和多组学分析（鸟枪宏基因组学，蛋白质组学，代表谢组学）。

可用服务：用珠子敲打的DNA提取

DNA浓度测量

对16S核糖体RNA基因（中等深度 - 即MiSeq-或高深度 - 即HiSeq）的V4区域进行测序，霰弹枪宏基因组测序

家庭粪便采样工具包

分装冷冻的粪便

处理粪便和灌洗样本的蛋白质组学

处理粪便和灌洗样本的代表谢组学

生物信息学核心

导演：珍妮弗 拉布斯博士（jlabus**[ta]**a.edu

生物信息学家：乔纳森 雅各布斯博士（JJacobs**[ta]**net.ucla.edu），Swapna乔希博士（SwapnaJoshi**[ta]**net.ucla.edu）

生物信息学核心为微生物核心产生的16S rRNA数据分析提供了一个标准化的管道。这包括alpha多样性，beta多样性，主坐标分析和差异丰度测试。生物信息学核心将安排一小时与生物信息学家协商审查结果。

定制数据分析可以与核心中的个体生物信息学家合作进行。可以采用以下

方法：多变量模型中差异分类群丰度

微生物共生网络

微生物模块

分层聚类

无监督分类

监督分类

预测的宏基因组学多元一体化途径分析

双方和三方网络

Procrustes叠加的数据集

日期：2018年1月26日，星期五

时间：12:00 - 1:00 PM

BSRB

演讲嘉宾：Grace Hancock和Nathanael Bangayan

加州大学洛杉矶分校健康科学媒体关系名录

Peter M.Bracke

传播总监

办公室：（310）206-4

手机：（323）599-1

PBracke**[ta]**net.ucla.edu

职员名册

艾米 艾尔宾310-267-***

aalbin**[ta]**net.ucla.edu

Brianna Aldrich310-206-***

bdeane**[ta]**tistry.ucla.edu

泰德 布劳恩310-319-***

tbraun**[ta]**net.ucla.edu

塔米 丹尼斯310-267-***

tdennis**[ta]**net.ucla.edu

菲尔汉普顿310-267-***

phampton**[ta]**net.ucla.edu

Ryan Hatoum310-267-***

rhatoum**[ta]**net.ucla.edu

Leigh跳跃者310-267-***

Lhopper**[ta]**net.ucla.edu

大卫奥尔莫斯310-267-***

dolmos**[ta]**net.ucla.edu

恩里克里维罗310-267-***

erivero**[ta]**net.ucla.edu

Elaine Schmidt310-267-***

eschmidt**[ta]**net.ucla.edu

西米?310-319-***

ssinger**[ta]**net.ucla.edu

按字母顺序跳动

成瘾医学诊所 - Leigh Hopper

管理，大卫Geffen医学院 - Tami丹尼斯

罗纳德 里根加州大学洛杉矶分校医疗中心Tami Dennis

艾滋病Enrique Rivero

过敏和免疫学Simi Singer

老年痴呆症护理中心即临床Enrique Rivero

老年痴呆症研究David Olmos

麻醉学Simi Singer

自闭症研究和治疗Leigh Hopper

减肥手术 - Ryan Hatoum

生物化学 - Elaine Schmidt

血液与血小板中心 - Elaine Schmidt

Blum拉美贫困与健康中心 - Enrique Rivero

大脑活动与结构 - Leigh Hopper

乳房中心（加州大学洛杉矶分校圣莫尼卡分校

Simi Singer

业务关系和伙伴关系 - 丹尼斯